pH homeostasis · Biochemistry

Acid-Base Balance MCQ

The Henderson-Hasselbalch relationship, respiratory and metabolic acidosis and alkalosis, and the chair-side scenarios from enamel pH to diabetic ketoacidosis. 25 MCQs and 4 INBDE patient cases.

25 practice MCQsQuick-reference tableMnemonics + clinical pearlsFull distractor explanations
High-yield review

Concept summary and clinical relevance.

Quick-reference structure first, then detailed coverage. Mnemonics in amber, clinical pearls in blue.

Acid-base balance keeps blood pH near 7.4 using a fast lever (the lungs, adjusting CO2) and a slow lever (the kidneys, adjusting bicarbonate). For dentistry the payoff is recognizing the chair-side events: the anxious hyperventilating patient, the over-sedated patient retaining CO2, and the systemically ill patient in diabetic ketoacidosis. Learn the four basic disturbances and the body's compensations, and these become quick to read.

The four primary acid-base disturbances
DisturbancePrimary changeCommon causeCompensation
Respiratory acidosisHigh CO2Hypoventilation (opioids, COPD)Kidney retains bicarbonate
Respiratory alkalosisLow CO2Hyperventilation (anxiety, pain)Kidney excretes bicarbonate
Metabolic acidosisLow bicarbonateAdded acid (DKA) or bicarbonate loss (diarrhea)Lungs blow off CO2 (Kussmaul)
Metabolic alkalosisHigh bicarbonateAcid loss (vomiting)Lungs retain CO2 (hypoventilation)

Henderson-Hasselbalch & Buffers

  • Normal arterial blood pH is tightly held between 7.35 and 7.45; below 7.35 is acidemia and above 7.45 is alkalemia.
  • The bicarbonate buffer is the main one: CO2 plus water forms carbonic acid, which dissociates into hydrogen ion and bicarbonate (carbonic anhydrase speeds the first step).
  • The Henderson-Hasselbalch relationship says pH tracks the ratio of bicarbonate to CO2; the lungs set CO2 (in minutes) and the kidneys set bicarbonate (over hours to days).
  • Phosphate and proteins (including hemoglobin) also buffer, but bicarbonate is the dominant extracellular buffer.

Respiratory Acidosis & Alkalosis

  • Respiratory acidosis is a high CO2 from hypoventilation: opioids and sedatives depressing the brainstem, COPD, or airway obstruction. The kidney compensates by retaining bicarbonate (slowly).
  • Respiratory alkalosis is a low CO2 from hyperventilation: anxiety, pain, hypoxia, or high altitude.
  • Hyperventilation lowers ionized calcium (more calcium binds albumin as pH rises), producing perioral and finger tingling and even carpopedal spasm.
  • A patient with COPD who chronically retains CO2 shows a compensated picture with a high bicarbonate on labs.
Clinical pearl, The hyperventilating patient: do not reach for oxygen
Acute anxiety hyperventilation in the chair causes respiratory alkalosis with perioral and finger tingling. The patient is not hypoxic, so supplemental oxygen does not help. Calm them, coach slow breathing, and have them rebreathe into cupped hands or a bag to raise CO2. Reserve oxygen for genuine hypoxia (for example, an asthma attack).

Metabolic Acidosis & Alkalosis

  • Metabolic acidosis is a low bicarbonate, from either added acid (diabetic ketoacidosis, lactic acidosis, salicylates, renal failure) or bicarbonate loss (diarrhea).
  • The anion gap (sodium minus chloride and bicarbonate) separates high-gap acidosis (added acids) from normal-gap acidosis (bicarbonate loss, as in diarrhea).
  • The lungs compensate for metabolic acidosis by deep, rapid Kussmaul breathing to blow off CO2.
  • Metabolic alkalosis is a high bicarbonate, classically from vomiting (loss of gastric acid), producing a hypochloremic, hypokalemic alkalosis.
Mnemonic, Vomiting vs diarrhea
“Vomiting is alkalosis, diarrhea is acidosis.” Vomiting loses acid (raising bicarbonate); diarrhea loses bicarbonate-rich fluid (lowering it).

Clinical Scenarios

  • Diabetic ketoacidosis: a high anion gap metabolic acidosis with fruity (acetone) breath and Kussmaul breathing, a medical emergency.
  • Anxiety hyperventilation: respiratory alkalosis with tingling and lightheadedness, managed by slow breathing, not oxygen.
  • Opioid over-sedation: respiratory acidosis with somnolence, slow breathing, and pinpoint pupils, reversed by naloxone with ventilatory support.
  • Salicylate (aspirin) overdose: a mixed picture of respiratory alkalosis and high anion gap metabolic acidosis, with tinnitus, important when patients self-medicate dental pain.
Clinical pearl, Why this matters in dentistry
The same chemistry runs from the enamel surface, where plaque acid below the critical pH near 5.5 demineralizes, to the bloodstream, where the body defends pH near 7.4. Chair-side, the acid-base events you must recognize are the hyperventilating anxious patient, the over-sedated patient retaining CO2, and the systemically ill patient (ketoacidosis, salicylate toxicity) who needs emergency referral.
Core Recall Check

25 board-style MCQs.

Active recall is the highest-yield study method. Pick an answer, check it, and read why every distractor is wrong.

0 of 25 answered · 0 correct
  1. Question 1
    Easy
    Normal arterial blood pH is maintained in the range of:
  2. Question 2
    Moderate
    The main buffer system of the blood is the:
  3. Question 3
    Moderate
    In the bicarbonate buffer, carbonic anhydrase speeds the reaction between carbon dioxide and:
  4. Question 4
    Moderate
    Blood pH depends on the ratio of bicarbonate, controlled by the kidneys, to carbon dioxide, controlled by the:
  5. Question 5
    Easy
    A blood pH of 7.30 is best described as:
  6. Question 6
    Moderate
    The lungs adjust blood pH faster than the kidneys because they regulate:
  7. Question 7
    Moderate
    Respiratory acidosis is caused by:
  8. Question 8
    Moderate
    Respiratory alkalosis in the dental chair is most often caused by:
  9. Question 9
    Hard
    The body compensates for a respiratory acid-base disturbance through the:
  10. Question 10
    Hard
    The perioral and finger tingling of acute hyperventilation is due to:
  11. Question 11
    Hard
    A patient with COPD who chronically retains CO2 typically shows a compensatory:
  12. Question 12
    Moderate
    Opioids cause respiratory acidosis by:
  13. Question 13
    Moderate
    Metabolic acidosis is defined by a primary decrease in:
  14. Question 14
    Hard
    Which causes a HIGH anion gap metabolic acidosis?
  15. Question 15
    Hard
    Severe diarrhea causes which acid-base disturbance?
  16. Question 16
    Moderate
    The respiratory compensation for a metabolic acidosis is:
  17. Question 17
    Moderate
    Prolonged vomiting characteristically causes:
  18. Question 18
    Hard
    The anion gap is calculated as sodium minus:
  19. Question 19
    Moderate
    Metabolic alkalosis is defined by a primary increase in:
  20. Question 20
    Moderate
    Diabetic ketoacidosis produces which acid-base picture?
  21. Question 21
    Moderate
    An anxious patient who is hyperventilating, lightheaded, and tingling but not hypoxic should be managed by:
  22. Question 22
    Moderate
    An over-sedated patient who is somnolent with slow, shallow breathing and pinpoint pupils most likely has:
  23. Question 23
    Hard
    Salicylate (aspirin) overdose classically produces:
  24. Question 24
    Moderate
    An early, characteristic symptom of salicylate toxicity is:
  25. Question 25
    Easy
    At the enamel surface, demineralization begins once plaque pH falls below the critical level of about:

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Clinical Reasoning Cases

INBDE patient cases.

4 ADA INBDE-format patient cases on acid-base balance. Each case is a shared patient box plus linked questions with full distractor explanations.

INBDE Patient Cases
Acid-Base Balance INBDE Patient Cases →

4 patient cases · 20 linked questions

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Author
Dr. Isaac Sun, DDS

Founder, KYT Dental Services. These MCQs are reviewed by a practicing clinician and offered as an educational reference for dental students.

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